Glossary entry (derived from question below)
English term or phrase:
colorectal cancer pathway
Hungarian translation:
colorectalis malignisatiós folyamat
Added to glossary by
Virág-Lilla Rácz
Apr 27, 2013 16:38
11 yrs ago
2 viewers *
English term
Dorottya Halvax
in colorectal cancer pathways
English to Hungarian
Medical
Medical (general)
With the publication of international CT colonography standards, which aim to improve quality of examinations, this review informs radiologists about the significance of flat polyps (adenomas and hyperplastic polyps) in colorectal cancer pathways. We describe flat polyp classification systems and propose how flat polyps should be reported to ensure patient management strategies are based on polyp morphology as well as size.
Proposed translations
(Hungarian)
| 4 +3 | colorectalis malignizációs folyamatokban |
Virág-Lilla Rácz
|
| 5 | colorectalis/kolorektális rák kialakulásának útvonalaiban |
Katalin Szilárd
|
Change log
May 11, 2013 19:29: Virág-Lilla Rácz Created KOG entry
Proposed translations
+3
43 mins
Virág-Lilla Rácz
Native in Hungarian
Specializes in field
30 years experience, medical translation
Native in: Hungarian 
Works in: English to Hungarian, English to Romanian, Romanian to Hungarian, and 3 more.
Related KudoZ points
:
44
Selected
colorectalis malignizációs folyamatokban
A vastagbél és végbél polip rosszindulatúvá válásáról van szó.
Note from asker:
| Köszönöm |
4 KudoZ points awarded for this answer.
Comment: "Selected automatically based on peer agreement."
4 hrs
Katalin Szilárd
Hungary
Hungary
Native in Hungarian
Specializes in field
Hungary
[email protected] MEDICAL4ENDCLIENTS
Native in: Hungarian
Works in: English to Hungarian, Hungarian to English
Related KudoZ points
:
345
colorectalis/kolorektális rák kialakulásának útvonalaiban
Itt a patways szó szerint útvonalakat jelent.
Lásd a referenciákat:
http://www.elsevierdirect.com/companions/9780123744197/exerc...
Colorectal cancer pathways: histopathology and molecular correlates. Panels A and B: The serrated pathway. Adenocarcinomas that develop through the serrated pathway arise from serrated polyps that include traditional serrated adenomas. (A) and sessile serrated adenomas (B) (H&E stain, original magnification 5× and 10×, for panels A and B, respectively). Traditional serrated adenomas show both serrated architecture and dysplasia similar to that seen in adenomatous mucosa, whereas sessile serrated adenomas reveal architectural abnormalities but no evidence of classic dysplasia. Panels C and D: The microsatellite instability pathway. Poorly differentiated colonic adenocarcinoma with prominent intratumoral lymphocytes, best seen in the inset (C) (H&E stain, original magnification 10×). By immunohistochemistry, the tumor cells are negative for MSH2, while the surrounding lymphocytes and stromal cells show preserved expression of MSH2 protein in the non-neoplastic cell nuclei (D) immunohistochemistry, original magnification 10×). (E) The presence of microsatellite instability in the tumor DNA is demonstrated by microsatellite instability at the microsatellite markers BAT25 and BAT26 characterized by the appearance of new PCR amplification peaks of smaller size (tailed arrows) as compared to non-neoplastic DNA from the same patient (arrow tip
http://ganymedes.lib.unideb.hu:8080/dea/bitstream/2437/89315...
7. oldal
Mikroszatellita instabilitás, avagy gén szintű instabilitás kialakulásával járó útvonalat.
Lásd a referenciákat:
http://www.elsevierdirect.com/companions/9780123744197/exerc...
Colorectal cancer pathways: histopathology and molecular correlates. Panels A and B: The serrated pathway. Adenocarcinomas that develop through the serrated pathway arise from serrated polyps that include traditional serrated adenomas. (A) and sessile serrated adenomas (B) (H&E stain, original magnification 5× and 10×, for panels A and B, respectively). Traditional serrated adenomas show both serrated architecture and dysplasia similar to that seen in adenomatous mucosa, whereas sessile serrated adenomas reveal architectural abnormalities but no evidence of classic dysplasia. Panels C and D: The microsatellite instability pathway. Poorly differentiated colonic adenocarcinoma with prominent intratumoral lymphocytes, best seen in the inset (C) (H&E stain, original magnification 10×). By immunohistochemistry, the tumor cells are negative for MSH2, while the surrounding lymphocytes and stromal cells show preserved expression of MSH2 protein in the non-neoplastic cell nuclei (D) immunohistochemistry, original magnification 10×). (E) The presence of microsatellite instability in the tumor DNA is demonstrated by microsatellite instability at the microsatellite markers BAT25 and BAT26 characterized by the appearance of new PCR amplification peaks of smaller size (tailed arrows) as compared to non-neoplastic DNA from the same patient (arrow tip
http://ganymedes.lib.unideb.hu:8080/dea/bitstream/2437/89315...
7. oldal
Mikroszatellita instabilitás, avagy gén szintű instabilitás kialakulásával járó útvonalat.
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